The main mineral that makes the bones strong and strong is calcium. The calcium content in bone and bone density gradually increases up to 30 years of age, when they reach their maximum. After that, the mineral density gradually decreases and it is necessary to provide sufficient nutrients in order to maintain the bone marrow system even in the mature age.
Osteoporosis is a disorder of bone loss. It is characterized by decreased bone density, disturbed bone structure, altered species and amount of bone protein. Osteoporosis leads to an increased risk of bone fractures, usually the hip and spinal vertebrae, or some other bone suffering from high loads.
Osteoporosis is considered a “quiet disease” because there are no noticeable symptoms until a bone fracture occurs.
With osteoporosis, the third-born or elderly population is most often confronted. Although it is more common in women, especially in menopause, osteoporosis affects both men over 50 years.
The highest risk is exposed to women, especially those with low body weight. High-risk groups include smokers, people who over-use alcohol, people with fracture history, and some diseases, including rheumatoid arthritis, hyperthyroidism, diabetes, and parathyroid gland diseases.
The latest research has fully illuminated the bone building mechanism: the protein responsible for the insertion of calcium into the bone, the so-called. Osteocalcin is the one that unlocks bones for calcium entry. However, after synthesis, osteocalcin is inactive and only in the presence of vitamin K2 is active and can perform its function. In other words, without vitamin K2, calcium remains unavailable to our bones even though we have entered it sufficiently.
Numerous clinical trials confirmed this: vitamin K2 completely prevents bone loss, while in some cases it even increases bone mass in people with osteoporosis. The evidence collected from seven Japanese studies shows that vitamin K2 supplements lead to a reduction in spinal fracture spleen of 60 percent, as well as a reduction in hip fracture and other non-vertebral fractures of 8 percent. Other studies have shown that vitamin K2 supplementation allows more efficient use of vitamin D and reduces the incidence of osteoporosis up to 25 percent.
Bone fracture occurs when the effect of an external force exceeds the density and bone strength. Breaks are usually the result of pressure or fall. Regardless of the cause of the fracture, broken bone significantly affects the quality of life, frequent pain, long-term immobilization and loss of independence.
Breaks are a common problem in people with osteopenia and osteoporosis. Osteoporotic fractures have a high mortality: over 20 percent of patients with a hip fracture die in the first year after the fracture, and most of them never fully recover.
Spasms of spinal vertebrae are also common and can occur spontaneously or after a slight injury. These are the so-called compressive fractures when chronic pain and deformation of the spinal column occur. Pain usually occurs suddenly, that part of the back becomes painful to the touch, after which problems with mobility, tension in the muscles and feeling in the hands can arise. People with spontaneous fractures of the spleen lose their altitude, and in time, it can also create humpiness.
Osteoporosis can also occur as a result of other diseases and drugs. This is the secondary secondary osteoporosis. The cause of reduced bone density can be chronic kidney disease or endocrine disease (thyroid disease, diabetes), chronic inflammatory bowel disease, poor nutrition, in obese and anorexic individuals.
Patients who are forced into long-term sleep due to poor health are also at great risk of bone density and increase the risk of fractures.
Many drugs can have a negative effect on mineral bone density and lead to osteoporosis. Corticosteroids used in the treatment of various diseases such as rheumatoid arthritis, multiple sclerosis, asthma, and others have a negative effect on the development of bone building cells. Some medicines used to treat epilepsy also prevent prolonged bone mineralization.